ะัะปะธ ะั ะตัั ะฝะต ะทะฝะฐะตัะต, ะบัะพ ัะฐะบะธะต ะะ, ะธ ะะฐะผ ะธะฝัะตัะตัะฝะพ - ะธััะพัะธั ะดะปั ะะฐั.
Duchenne muscular dystrophy (DMD) is a rare genetic disorder characterized by the progressive weakening and wasting of muscles, affecting skeletal, smooth, and cardiac muscle tissues. The underlying cause of muscle damage is the absence of the sarcolemmal protein dystrophin due to variations in the DMD gene located on Xp21.2. Symptoms of DMD typically manifest in early childhood, with affected boys often experiencing delayed walking, typically after 18 months, along with speech and developmental delays. Individuals with DMD may also face autism and behavioral issues, including ADHD, anxiety, and obsessive-compulsive disorder. Unfortunately, untreated children with DMD rarely achieve the ability to run or jump. The condition progresses rapidly, leading to a characteristic waddling gait and a positive Gowers' sign. Climbing stairs becomes challenging, and frequent falls occur. Independent walking is typically lost between the ages of 6 and 13, with an average age of 9.5 in non-steroid treated cases. Once ambulation is lost, joint contractures and scoliosis develop rapidly, and untreated patients typically succumb to respiratory failure or cardiomyopathy in their late teens to early twenties. Diagnosis of DMD is suspected based on clinical presentation, family history, and laboratory findings, particularly elevated serum creatine kinase (CK) levels, often 100-200 times the normal range. Genetic testing is the definitive method and involves multiplex-ligation dependent probe amplification (MLPA) for detecting exon deletions and duplications and full gene sequencing to identify small deletions, duplications, non-sense mutations, or point mutations. Differential diagnoses include severe Becker muscular dystrophy and limb-girdle muscular dystrophy. The gold standard treatment for DMD involves corticosteroids such as prednisolone, prednisone, or deflazacort.
hello ! not giving credits is alright by the writer of this book ^^highest ranks : #1 - aestheticusername#1 - kpopusername#4 - usernames#5 - username#8 - animeusernamesโ ๏ธplease do not copy & claim it as yours(this book has finally come to an end :), no more updates ^_^)might make another ,,
"Whats one of your dreams? One thing you really want?"Well i wanna be loved"Well I'm here, you all are real. It already came true"I wanna be told I'm more then enough"Oh really? Thats it? nothing else?"I want that voice in the back of my head to go away. To know I'm not a disappointment, a wast of space, that someone does want me."Nope, as long as your here, as long as everyone is here I'm happy. This is a dream come true"All (y/n) wants is to go into the my hero academia world, meet the characters and feel loved. well what happens when running away from her abusive house she finds her self falling in the ski?uh read at your own risk of bad writing. this is a tokoyami x reader CAUSE MR UNDERRATED PRETTY BOY HERE NEEDS SOME MORE LOVEif you do read this then thank you and please tell me what you think of it
Neville and Hermione would make a brilliant couple. If you're not convinced, here's a collection of short stories to change your mind.
๐๐๐๐๐๐๐๐ ๐๐๐๐๐๐๐๐๐ ๐๐ ๐กโ๐ ๐๐๐ข๐โ๐ก๐๐ ๐๐ ๐พ๐๐๐ ๐ด๐๐กโ๐ข๐. ๐โ๐ ๐๐โ๐๐๐๐ก๐๐ ๐๐๐๐๐๐๐'๐ ๐๐๐ค๐๐, โ๐๐ค๐๐ฃ๐๐, ๐กโ๐ ๐๐๐๐๐๐๐ ๐ โ๐๐๐ ๐๐๐, ๐๐๐๐ ๐๐ ๐๐๐ฃ๐ ๐ค๐๐กโ ๐๐๐๐๐๐'๐ ๐ด๐๐๐๐๐๐ก๐๐๐. ๐โ๐ ๐๐๐๐ ๐ค๐๐กโ๐๐ข๐ก ๐๐๐๐ฃ๐๐๐ ๐ ๐ก๐๐๐๐, ๐๐๐๐๐๐๐๐ ๐กโ๐ ๐ค๐๐ง๐๐๐๐ โ๐๐๐๐ก. ๐ถ๐๐๐ก๐ข๐๐๐๐ ๐๐๐ก๐๐, ๐ โ๐ ๐๐๐ข๐๐๐ก๐๐ ๐ค๐๐กโ โ๐๐, ๐๐ข๐ก ๐ค๐๐ข๐๐ ๐กโ๐๐๐๐ ๐๐ ๐กโ๐ ๐ ๐๐๐? DISCLAIMERโ๏ธโ๏ธโ๏ธ I DO NOT OWN COPYRIGHT OF TALES OF ARCADIA OR THE PICTURES!! ALL GOES TO RIGHTFUL OWNER!!!!*UNDER EDITING*๐๐๐ง๐ค๐๐ #6 ๐ข๐ง ๐ฃ๐ข๐ฆ๐ฅ๐๐ค๐ ๐๐ฌ ๐จ๐ 3/25/21 ๐๐๐ง๐ค๐๐ #11 ๐ข๐ง ๐๐ฅ๐๐ข๐ซ๐๐ง๐ฎ๐ง๐๐ณ ๐๐ฌ ๐จ๐ 4/8/21๐๐๐ง๐ค๐๐ #13 ๐ข๐ง ๐ฐ๐ข๐ณ๐๐ซ๐๐ฌ๐ญ๐๐ฅ๐๐ฌ๐จ๐๐๐ซ๐๐๐๐ข๐ ๐๐ฌ ๐จ๐ 4/13/21
I'm gonna make some headcanons about the disventure camp characters because i'm bored. I don't own the disventure camp characters, they belong to oddnationcartoons.